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- Insulin…an Undeserved Bad Reputation
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- The Pitfalls of Body Fat “Measurement”, Part 3: Bod Pod
- The Pitfalls of Body Fat “Measurement”, Part 5: Skinfolds
- The Pitfalls of Body Fat “Measurement”, Part 6: Dual-Energy X-Ray Absorptiometry (DEXA)
- The Pitfalls of Body Fat “Measurement”, The Final Chapter
- The Pitfalls of Body Fat “Measurement”: Part 1
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- How Much Do Variations in Physiological Testosterone Matter To Your Gains?
- Periodization / Variation in Repetition Ranges for Muscle Size: Your Complete Evidence Based Guide
- Set Volume for Muscle Size: The Ultimate Evidence Based Bible
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- Post-Exercise Energy Expenditure, Part 4: The Effect of Low Volume Weight Training
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- Why Do Meal Replacements Work?
I’m a little confused when you say ” In fact, when you knock out the insulin receptor in mice so that insulin cannot stimulate glucose uptake into muscle or fat cells (yet you keep the insulin receptor intact on other cells like brain and liver), the animals do not become diabetic and they have normal blood sugars.” Do you mind elaborating a little bit more on this? I’m guessing that by decreasing the amount of insulin receptors in mice you would expect their blood sugar to go up but this was not the case. Please correct me if I’m wrong